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  • The main molecular mechanisms involved in oxaliplatin resistance appears to depend upon: (i) decreased cellular accumulation, which is thought to be related to a greater activity of the ATP7B exporter rather than to the activity of P-glycoprotein (P-gp) and multidrug resistance protein 1 (MRP1), and (ii) more efficient repair of oxaliplatin-induced DNA-damage by NER (Nucleotide Excision Repair) [17].
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