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  • Previous reports have indicated that sorafenib treatment stimulates SHP-1 and/or SHP-2 tyrosine phosphatase activity in breast cancer [32], glioblastoma [30], HCC [14] and pancreatic cancer [31] and that orthovanadate reduces the sorafenib-induced inhibition of signal transducer and activator of transcription 3 (STAT3)- mediated tyrosine phosphorylation and increased PTP and SHP-2 activity in glioma cells [30].
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