M-CSF-stimulated CD11c expression in BMM was reduced by inhibition of PLCĪ³2, Akt, and ERK, whereas the absence of MCP-1 abolished this reduction, suggesting that the ability of MCP-1 to induce CD11c expression is via activation of PLCĪ³2, Akt, and ERK in BMM.
![[This page as RDF]](http://data.gesis.org/somesci/static/rdf-icon.gif){kind=icon}